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Nonmigraine-associated TRESK K(+) channel variant C110R does not increase the excitability of trigeminal ganglion neurons
Recent genetic studies suggest that dysfunction of ion channels and transporters may contribute to migraine pathophysiology. A migraine-associated frameshift mutation in the TWIK-related spinal cord K(+) (TRESK) channel results in nonfunctional channels. Moreover, mutant TRESK subunits exert a domin...
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| Main Authors: | , , , |
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| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
American Physiological Society
2014
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4122697/ https://ncbi.nlm.nih.gov/pubmed/24805079 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/jn.00267.2014 |
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