Endoplasmic reticulum: ER stress regulates mitochondrial bioenergetics

Endoplasmic reticulum (ER) stress activates an adaptive unfolded protein response (UPR) that facilitates cellular repair, however, under prolonged ER stress, the UPR can ultimately trigger apoptosis thereby terminating damaged cells. The molecular mechanisms responsible for execution of the cell dea...

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Bibliografische gegevens
Hoofdauteurs: Bravo, Roberto, Gutierrez, Tomás, Paredes, Felipe, Gatica, Damián, Rodriguez, Andrea E., Pedrozo, Zully, Chiong, Mario, Parra, Valentina, Quest, Andrew F.G., Rothermel, Beverly A., Lavandero, Sergio
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2011
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Article
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4118286/
https://ncbi.nlm.nih.gov/pubmed/22064245
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.biocel.2011.10.012
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