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Endoplasmic reticulum: ER stress regulates mitochondrial bioenergetics

Endoplasmic reticulum (ER) stress activates an adaptive unfolded protein response (UPR) that facilitates cellular repair, however, under prolonged ER stress, the UPR can ultimately trigger apoptosis thereby terminating damaged cells. The molecular mechanisms responsible for execution of the cell dea...

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Dettagli Bibliografici
Autori principali: Bravo, Roberto, Gutierrez, Tomás, Paredes, Felipe, Gatica, Damián, Rodriguez, Andrea E., Pedrozo, Zully, Chiong, Mario, Parra, Valentina, Quest, Andrew F.G., Rothermel, Beverly A., Lavandero, Sergio
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2011
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4118286/
https://ncbi.nlm.nih.gov/pubmed/22064245
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.biocel.2011.10.012
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