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Altered Inhibition of Cx26 Hemichannels by pH and Zn(2+) in the A40V Mutation Associated with Keratitis-Ichthyosis-Deafness Syndrome
Excessive opening of undocked Cx26 hemichannels in the plasma membrane is associated with disease pathogenesis in keratitis-ichthyosis-deafness (KID) syndrome. Thus far, excessive opening of KID mutant hemichannels has been attributed, almost solely, to aberrant inhibition by extracellular Ca(2+). T...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
American Society for Biochemistry and Molecular Biology
2014
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4118113/ https://ncbi.nlm.nih.gov/pubmed/24939841 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M114.578757 |
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