NAC Attenuates LPS-Induced Toxicity in Aspirin-Sensitized Mouse Macrophages via Suppression of Oxidative Stress and Mitochondrial Dysfunction

Lignende værker

• NAC attenuates LPS-induced toxicity in aspirin-sensitized mouse macrophages via suppression of oxidative stress and mitochondrial dysfunction.
  af: Haider Raza, et al.
  Udgivet: (2014-01-01)
• Potentiation of LPS-Induced Apoptotic Cell Death in Human Hepatoma HepG2 Cells by Aspirin via ROS and Mitochondrial Dysfunction: Protection by N-Acetyl Cysteine.
  af: Haider Raza, et al.
  Udgivet: (2016-01-01)
• Potentiation of LPS-Induced Apoptotic Cell Death in Human Hepatoma HepG2 Cells by Aspirin via ROS and Mitochondrial Dysfunction: Protection by N-Acetyl Cysteine
  af: Raza, Haider, et al.
  Udgivet: (2016)
• Implications of Altered Glutathione Metabolism in Aspirin-Induced Oxidative Stress and Mitochondrial Dysfunction in HepG2 Cells
  af: Raza, Haider, et al.
  Udgivet: (2012)
• Exercise‐induced alterations in pancreatic oxidative stress and mitochondrial function in type 2 diabetic Goto‐Kakizaki rats
  af: Raza, Haider, et al.
  Udgivet: (2016)