Extrasynaptic glutamate release through cystine/glutamate antiporter contributes to ischemic damage

During brain ischemia, an excessive release of glutamate triggers neuronal death through the overactivation of NMDA receptors (NMDARs); however, the underlying pathways that alter glutamate homeostasis and whether synaptic or extrasynaptic sites are responsible for excess glutamate remain controvers...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
मुख्य लेखकों: Soria, Federico N., Pérez-Samartín, Alberto, Martin, Abraham, Gona, Kiran Babu, Llop, Jordi, Szczupak, Boguslaw, Chara, Juan Carlos, Matute, Carlos, Domercq, María
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: American Society for Clinical Investigation 2014
विषय:
Research Article
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC4109556/
https://ncbi.nlm.nih.gov/pubmed/25036707
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI71886
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