The Nuclear Receptor Peroxisome Proliferator-activated Receptor-β/δ (PPARβ/δ) Promotes Oncogene-induced Cellular Senescence through Repression of Endoplasmic Reticulum Stress

Endoplasmic reticulum (ER) stress and ER stress-associated unfolded protein response (UPR) can promote cancer cell survival, but it remains unclear whether they can influence oncogene-induced senescence. The present study examined the role of ER stress in senescence using oncogene-dependent models....

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Main Authors: Zhu, Bokai, Ferry, Christina H., Markell, Lauren K., Blazanin, Nicholas, Glick, Adam B., Gonzalez, Frank J., Peters, Jeffrey M.
Formáid: Artigo
Teanga:Inglês
Foilsithe: American Society for Biochemistry and Molecular Biology 2014
Ábhair:
Molecular Bases of Disease
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC4106326/
https://ncbi.nlm.nih.gov/pubmed/24898257
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M114.551069
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