Monoclonal antibodies raised against Guillain-Barré syndrome–associated Campylobacter jejuni lipopolysaccharides react with neuronal gangliosides and paralyze muscle-nerve preparations

Guillain-Barré syndrome and its variant, Miller-Fisher syndrome, are acute, postinfectious, autoimmune neuropathies that frequently follow Campylobacter jejuni enteritis. The pathogenesis is believed to involve molecular mimicry between sialylated epitopes on C. jejuni LPSs and neural gangliosides....

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Detaylı Bibliyografya
Asıl Yazarlar: Goodyear, Carl S., O’Hanlon, Graham M., Plomp, Jaap J., Wagner, Eric R., Morrison, Ian, Veitch, Jean, Cochrane, Lynne, Bullens, Roland W. M., Molenaar, Peter C., Conner, Joe, Willison, Hugh J.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Clinical Investigation 1999
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Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC408431/
https://ncbi.nlm.nih.gov/pubmed/10491405
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