Protein 4.1R–deficient mice are viable but have erythroid membrane skeleton abnormalities
A diverse family of protein 4.1R isoforms is encoded by a complex gene on human chromosome 1. Although the prototypical 80-kDa 4.1R in mature erythrocytes is a key component of the erythroid membrane skeleton that regulates erythrocyte morphology and mechanical stability, little is known about 4.1R...
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| Udgivet i: | J Clin Invest |
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| Principais autores: | , , , , , , , , , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
American Society for Clinical Investigation
1999
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC407893/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9927493/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI3858 |
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