Protein 4.1R–deficient mice are viable but have erythroid membrane skeleton abnormalities

A diverse family of protein 4.1R isoforms is encoded by a complex gene on human chromosome 1. Although the prototypical 80-kDa 4.1R in mature erythrocytes is a key component of the erythroid membrane skeleton that regulates erythrocyte morphology and mechanical stability, little is known about 4.1R...

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Hlavní autoři: Shi, Zheng-Tao, Afzal, Veena, Coller, Barry, Patel, Dipti, Chasis, Joel A., Parra, Marilyn, Lee, Gloria, Paszty, Chris, Stevens, Mary, Walensky, Loren, Peters, Luanne L., Mohandas, Narla, Rubin, Edward, Conboy, John G.
Médium: Artigo
Jazyk:Inglês
Vydáno: American Society for Clinical Investigation 1999
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC407893/
https://ncbi.nlm.nih.gov/pubmed/9927493
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