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Distinct functional roles for the two SLX4 ubiquitin-binding UBZ domains mutated in Fanconi anemia
Defects in SLX4, a scaffold for DNA repair nucleases, result in Fanconi anemia (FA), due to the defective repair of inter-strand DNA crosslinks (ICLs). Some FA patients have an SLX4 deletion removing two tandem UBZ4-type ubiquitin-binding domains that are implicated in protein recruitment to sites o...
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| Main Authors: | , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The Company of Biologists
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4075355/ https://ncbi.nlm.nih.gov/pubmed/24794496 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/jcs.146167 |
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