טוען...
Resident fibroblast lineages mediate pressure overload–induced cardiac fibrosis
Activation and accumulation of cardiac fibroblasts, which result in excessive extracellular matrix deposition and consequent mechanical stiffness, myocyte uncoupling, and ischemia, are key contributors to heart failure progression. Recently, endothelial-to-mesenchymal transition (EndoMT) and the rec...
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| Main Authors: | , , , , , , , , , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Clinical Investigation
2014
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4071409/ https://ncbi.nlm.nih.gov/pubmed/24937432 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI74783 |
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