Resident fibroblast lineages mediate pressure overload–induced cardiac fibrosis

Activation and accumulation of cardiac fibroblasts, which result in excessive extracellular matrix deposition and consequent mechanical stiffness, myocyte uncoupling, and ischemia, are key contributors to heart failure progression. Recently, endothelial-to-mesenchymal transition (EndoMT) and the rec...

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Hlavní autoři: Moore-Morris, Thomas, Guimarães-Camboa, Nuno, Banerjee, Indroneal, Zambon, Alexander C., Kisseleva, Tatiana, Velayoudon, Aurélie, Stallcup, William B., Gu, Yusu, Dalton, Nancy D., Cedenilla, Marta, Gomez-Amaro, Rafael, Zhou, Bin, Brenner, David A., Peterson, Kirk L., Chen, Ju, Evans, Sylvia M.
Médium: Artigo
Jazyk:Inglês
Vydáno: American Society for Clinical Investigation 2014
Témata:
Research Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4071409/
https://ncbi.nlm.nih.gov/pubmed/24937432
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI74783
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