Resident fibroblast lineages mediate pressure overload–induced cardiac fibrosis

Activation and accumulation of cardiac fibroblasts, which result in excessive extracellular matrix deposition and consequent mechanical stiffness, myocyte uncoupling, and ischemia, are key contributors to heart failure progression. Recently, endothelial-to-mesenchymal transition (EndoMT) and the rec...

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Detalhes bibliográficos
Main Authors: Moore-Morris, Thomas, Guimarães-Camboa, Nuno, Banerjee, Indroneal, Zambon, Alexander C., Kisseleva, Tatiana, Velayoudon, Aurélie, Stallcup, William B., Gu, Yusu, Dalton, Nancy D., Cedenilla, Marta, Gomez-Amaro, Rafael, Zhou, Bin, Brenner, David A., Peterson, Kirk L., Chen, Ju, Evans, Sylvia M.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Clinical Investigation 2014
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4071409/
https://ncbi.nlm.nih.gov/pubmed/24937432
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI74783
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