Resident fibroblast lineages mediate pressure overload–induced cardiac fibrosis

Activation and accumulation of cardiac fibroblasts, which result in excessive extracellular matrix deposition and consequent mechanical stiffness, myocyte uncoupling, and ischemia, are key contributors to heart failure progression. Recently, endothelial-to-mesenchymal transition (EndoMT) and the rec...

Disgrifiad llawn

Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awduron: Moore-Morris, Thomas, Guimarães-Camboa, Nuno, Banerjee, Indroneal, Zambon, Alexander C., Kisseleva, Tatiana, Velayoudon, Aurélie, Stallcup, William B., Gu, Yusu, Dalton, Nancy D., Cedenilla, Marta, Gomez-Amaro, Rafael, Zhou, Bin, Brenner, David A., Peterson, Kirk L., Chen, Ju, Evans, Sylvia M.
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: American Society for Clinical Investigation 2014
Pynciau:
Research Article
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC4071409/
https://ncbi.nlm.nih.gov/pubmed/24937432
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI74783
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