IL-4 Signaling Drives a Unique Arginase(+)/IL-1β(+) Microglia Phenotype and Recruits Macrophages to the Inflammatory CNS: Consequences of Age-Related Deficits in IL-4Rα after Traumatic Spinal Cord Injury

Alternative activation of microglia/macrophages (M2a) by interleukin (IL)-4 is purported to support intrinsic growth and repair processes after CNS injury. Nonetheless, alternative activation of microglia is poorly understood in vivo, particularly in the context of inflammation, injury, and aging. H...

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Detaylı Bibliyografya
Asıl Yazarlar: Fenn, Ashley M., Hall, Jodie C.E., Gensel, John C., Popovich, Phillip G., Godbout, Jonathan P.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Society for Neuroscience 2014
Konular:
Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4069360/
https://ncbi.nlm.nih.gov/pubmed/24966389
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1146-14.2014
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