IL-4 Signaling Drives a Unique Arginase(+)/IL-1β(+) Microglia Phenotype and Recruits Macrophages to the Inflammatory CNS: Consequences of Age-Related Deficits in IL-4Rα after Traumatic Spinal Cord Injury

Alternative activation of microglia/macrophages (M2a) by interleukin (IL)-4 is purported to support intrinsic growth and repair processes after CNS injury. Nonetheless, alternative activation of microglia is poorly understood in vivo, particularly in the context of inflammation, injury, and aging. H...

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Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς: Fenn, Ashley M., Hall, Jodie C.E., Gensel, John C., Popovich, Phillip G., Godbout, Jonathan P.
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: Society for Neuroscience 2014
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Articles
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4069360/
https://ncbi.nlm.nih.gov/pubmed/24966389
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1146-14.2014
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