No Contribution of IP(3)-R(2) to Disease Phenotype in Models of Dilated Cardiomyopathy or Pressure Overload Hypertrophy

BACKGROUND: We investigated the contribution of inositol(1,4,5)trisphosphate (Ins(1,4,5)P(3), IP(3)) receptors (IP(3)-R) to disease progression in mouse models of dilated cardiomyopathy (DCM) and pressure overload hypertrophy. Mice expressing mammalian sterile 20 like kinase and dominant negative ph...

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Detalhes bibliográficos
Main Authors: Cooley, Nicola, Ouyang, Kunfu, McMullen, Julie R., Kiriazis, Helen, Sheikh, Farah, Wu, Wei, Mu, Yongxin, Du, Xiao-Jun, Chen, Ju, Woodcock, Elizabeth A.
Formato: Artigo
Idioma:Inglês
Publicado em: 2012
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4028972/
https://ncbi.nlm.nih.gov/pubmed/23258573
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCHEARTFAILURE.112.972158
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