Extensive dysregulations of oligodendrocytic and astrocytic connexins are associated with disease progression in an amyotrophic lateral sclerosis mouse model

BACKGROUND: Non-cell-autonomous motor neuronal death is suggested in a mutant Cu/Zn superoxide dismutase 1 (mSOD1)-mediated amyotrophic lateral sclerosis (ALS) model, in which glial cells play significant roles in disease progression. Connexins (Cxs) form homotypic or heterotypic gap junctions (GJs)...

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Detalhes bibliográficos
Main Authors: Cui, Yiwen, Masaki, Katsuhisa, Yamasaki, Ryo, Imamura, Shihoko, Suzuki, Satoshi O, Hayashi, Shintaro, Sato, Shinya, Nagara, Yuko, Kawamura, Mami F, Kira, Jun-ichi
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2014
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4016493/
https://ncbi.nlm.nih.gov/pubmed/24597481
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-11-42
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