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Leptin-stimulated K(ATP) channel trafficking: A new paradigm for β-cell stimulus-secretion coupling?

Insulin secretion from pancreatic β-cells is initiated by the closure of ATP-sensitive K(+) channels (K(ATP)) in response to high concentrations of glucose, and this action of glucose is counteracted by the hormone leptin, an adipokine that signals through the Ob-R(b) receptor to increase K(ATP) cha...

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Xehetasun bibliografikoak
Egile Nagusiak: Holz, George G, Chepurny, Oleg G, Leech, Colin A
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Landes Bioscience 2013
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC4010576/
https://ncbi.nlm.nih.gov/pubmed/24213304
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/isl.26958
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