Yeast upstream activator protein GCN4 can stimulate transcription when its binding site replaces the TATA element.

We replaced the required TATA element of a yeast gal-his3 promoter by a binding site for GCN4, a protein that normally activates transcription when bound upstream of a TATA element. Surprisingly, GCN4 efficiently activates his3 transcription from wild-type initiation sites, though in a pattern assoc...

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Veröffentlicht in:	EMBO J
Hauptverfasser:	Chen, W, Struhl, K
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	Nature Publishing Group 1989
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC400798/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2653813/ https://ncbi.nlm.nih.govhttps://doi.org/10.1002/j.1460-2075.1989.tb03372.x
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