Nrf2 activation-induced hepatic VLDLR overexpression in response to oxidative stress contributes to alcoholic liver disease in mice

Lignende værker

• Protection of Nicotinic Acid against Oxidative Stress-Induced Cell Death in Hepatocytes Contributes to Its Beneficial Effect on Alcohol-induced Liver Injury in Mice
  af: Dou, Xiaobing, et al.
  Udgivet: (2013)
• Inhibition of NF-κB Activation by 4-Hydroxynonenal Contributes to Liver Injury in a Mouse Model of Alcoholic Liver Disease
  af: Dou, Xiaobing, et al.
  Udgivet: (2012)
• Rectification of impaired adipose tissue methylation status and lipolytic response contributes to hepatoprotective effect of betaine in a mouse model of alcoholic liver disease
  af: Dou, Xiaobing, et al.
  Udgivet: (2014)
• Hepatic SIRT3 Upregulation in Response to Chronic Alcohol Consumption Contributes to Alcoholic Liver Disease in Mice
  af: Ma, Yue, et al.
  Udgivet: (2019)
• TLR9 Signaling Protects Alcohol-Induced Hepatic Oxidative Stress but Worsens Liver Inflammation in Mice
  af: Hao, Liuyi, et al.
  Udgivet: (2021)