PTEN knockdown alters dendritic spine/protrusion morphology, not density

Mutations in phosphatase and tensin homolog deleted on chromosome ten (PTEN) are implicated in neuropsychiatric disorders including autism. Previous studies report that PTEN knockdown in neurons in vivo leads to increased spine density and synaptic activity. To better characterize synaptic changes i...

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Detalhes bibliográficos
Main Authors: Haws, Michael E., Jaramillo, Thomas C., Espinosa-Becerra, Felipe, Widman, Allie, Stuber, Garret D., Sparta, Dennis R., Tye, Kay M., Russo, Scott J., Parada, Luis F., Kaplitt, Michael, Bonci, Antonello, Powell, Craig M.
Formato: Artigo
Idioma:Inglês
Publicado em: 2014
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3945606/
https://ncbi.nlm.nih.gov/pubmed/24264880
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/cne.23488
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