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Non-canonical p53 signaling to promote invasion

It has been known for a number of years that mutated “inactive” p53 proteins still capable of binding to DNA per se, can bind to DNA sequences that are non-canonical for p53, with for example, a resultant increase in the transcription and expression of growth factor receptors such as ERBB1,(1)(,)(2)...

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Detalhes bibliográficos
Autor principal: Dent, Paul
Formato: Artigo
Idioma:Inglês
Publicado em: Landes Bioscience 2013
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3926882/
https://ncbi.nlm.nih.gov/pubmed/24025254
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/cbt.26174
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