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Non-canonical p53 signaling to promote invasion
It has been known for a number of years that mutated “inactive” p53 proteins still capable of binding to DNA per se, can bind to DNA sequences that are non-canonical for p53, with for example, a resultant increase in the transcription and expression of growth factor receptors such as ERBB1,(1)(,)(2)...
Bewaard in:
| Hoofdauteur: | |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Landes Bioscience
2013
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3926882/ https://ncbi.nlm.nih.gov/pubmed/24025254 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/cbt.26174 |
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