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Blockage of the Upregulation of Voltage-Gated Sodium Channel Na(v)1.3 Improves Outcomes after Experimental Traumatic Brain Injury
Excessive active voltage-gated sodium channels are responsible for the cellular abnormalities associated with secondary brain injury following traumatic brain injury (TBI). We previously presented evidence that significant upregulation of Na(v)1.3 expression occurs in the rat cortex at 2 h and 12 h...
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| Główni autorzy: | , , , , , , |
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| Format: | Artigo |
| Język: | Inglês |
| Wydane: |
Mary Ann Liebert, Inc.
2014
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| Hasła przedmiotowe: | |
| Dostęp online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3922240/ https://ncbi.nlm.nih.gov/pubmed/24313291 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2013.2899 |
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