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Doxorubicin-induced carbonylation and degradation of cardiac myosin binding protein C promote cardiotoxicity
Dose-dependent oxidative stress by the anthracycline doxorubicin (Dox) and other chemotherapeutic agents causes irreversible cardiac damage, restricting their clinical effectiveness. We hypothesized that the resultant protein oxidation could be monitored and correlated with physiological functional...
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| Huvudupphovsmän: | , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
National Academy of Sciences
2014
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3918758/ https://ncbi.nlm.nih.gov/pubmed/24449919 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1321783111 |
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