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Reduction of endoplasmic reticulum stress attenuates the defects caused by Drosophila mitofusin depletion
Ablation of the mitochondrial fusion and endoplasmic reticulum (ER)–tethering protein Mfn2 causes ER stress, but whether this is just an epiphenomenon of mitochondrial dysfunction or a contributor to the phenotypes in mitofusin (Mfn)-depleted Drosophila melanogaster is unclear. In this paper, we sho...
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| 主要な著者: | , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
The Rockefeller University Press
2014
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3912536/ https://ncbi.nlm.nih.gov/pubmed/24469638 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.201306121 |
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