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Dexamethasone exacerbates cerebral edema and brain injury following lithium-pilocarpine induced status epilepticus()

Anti-inflammatory therapies are the current most plausible drug candidates for anti-epileptogenesis and neuroprotection following prolonged seizures. Given that vasogenic edema is widely considered to be detrimental for outcome following status epilepticus, the anti-inflammatory agent dexamethasone...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Duffy, B.A., Chun, K.P., Ma, D., Lythgoe, M.F., Scott, R.C.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Academic Press 2014
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3905166/
https://ncbi.nlm.nih.gov/pubmed/24333865
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2013.12.001
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