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Dexamethasone exacerbates cerebral edema and brain injury following lithium-pilocarpine induced status epilepticus()
Anti-inflammatory therapies are the current most plausible drug candidates for anti-epileptogenesis and neuroprotection following prolonged seizures. Given that vasogenic edema is widely considered to be detrimental for outcome following status epilepticus, the anti-inflammatory agent dexamethasone...
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| Autors principals: | , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Academic Press
2014
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3905166/ https://ncbi.nlm.nih.gov/pubmed/24333865 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2013.12.001 |
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