A point mutation in the HIV-1 Tat responsive element is associated with postintegration latency.

Study of the mechanism of HIV-1 postintegration latency in the ACH2 cell line demonstrates that these cells failed to increase HIV-1 production following treatment with exogenous Tat. Reasoning that the defect in ACH2 cells involves the Tat response, we analyzed the sequence of tat cDNA and Tat resp...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Emiliani, S, Van Lint, C, Fischle, W, Paras, P, Ott, M, Brady, J, Verdin, E
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 1996
Gaiak:
Research Article
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC39030/
https://ncbi.nlm.nih.gov/pubmed/8692823
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