A point mutation in the HIV-1 Tat responsive element is associated with postintegration latency.
Study of the mechanism of HIV-1 postintegration latency in the ACH2 cell line demonstrates that these cells failed to increase HIV-1 production following treatment with exogenous Tat. Reasoning that the defect in ACH2 cells involves the Tat response, we analyzed the sequence of tat cDNA and Tat resp...
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| Yayımlandı: | Proc Natl Acad Sci U S A |
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| Asıl Yazarlar: | , , , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
National Academy of Sciences
1996
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC39030/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8692823/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.93.13.6377 |
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