A point mutation in the HIV-1 Tat responsive element is associated with postintegration latency.

Study of the mechanism of HIV-1 postintegration latency in the ACH2 cell line demonstrates that these cells failed to increase HIV-1 production following treatment with exogenous Tat. Reasoning that the defect in ACH2 cells involves the Tat response, we analyzed the sequence of tat cDNA and Tat resp...

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Veröffentlicht in:	Proc Natl Acad Sci U S A
Hauptverfasser:	Emiliani, S, Van Lint, C, Fischle, W, Paras, P, Ott, M, Brady, J, Verdin, E
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	National Academy of Sciences 1996
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC39030/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8692823/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.93.13.6377
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