Microglial Receptor for Advanced Glycation End Product-Dependent Signal Pathway Drives β-Amyloid-Induced Synaptic Depression and Long-Term Depression Impairment in Entorhinal Cortex

Eitemau Tebyg

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• Receptor for Advanced Glycation End Product-Dependent Activation of p38 Mitogen-Activated Protein Kinase Contributes to Amyloid-β-Mediated Cortical Synaptic Dysfunction
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• An isoform-selective p38αMAPK inhibitor rescues early entorhinal cortex dysfunctions in a mouse model of Alzheimer’s disease
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