טוען...
Apo-states of calmodulin and CaBP1 control Ca(V)1 voltage-gated calcium channel function through direct competition for the IQ domain
In neurons, binding of calmodulin (CaM) or calcium-binding protein 1 (CaBP1) to the Ca(V)1 (L-type) voltage-gated calcium channel IQ domain endows the channel with diametrically opposed properties. CaM causes calcium-dependent inactivation (CDI) and limits calcium entry, whereas CaBP1 blocks CDI and...
שמור ב:
| Main Authors: | , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2013
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3839849/ https://ncbi.nlm.nih.gov/pubmed/23811053 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jmb.2013.06.024 |
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