Increased mTORC1 activity contributes to atherosclerosis in apolipoprotein E knockout mice and in vascular smooth muscle cells()

Samankaltaisia teoksia

• Platelet microparticles contribute to aortic vascular endothelial injury in diabetes via the mTORC1 pathway
  Tekijä: Wang, Gui-hua, et al.
  Julkaistu: (2018)
• Activation of renin-angiotensin system is involved in dyslipidemia-mediated renal injuries in apolipoprotein E knockout mice and HK-2 cells
  Tekijä: Ni, Jie, et al.
  Julkaistu: (2013)
• Txnip Ablation Reduces Vascular Smooth Muscle Cell Inflammation and Ameliorates Atherosclerosis in Apolipoprotein E Knockout Mice
  Tekijä: Byon, Chang Hyun, et al.
  Julkaistu: (2015)
• Inflammatory Stress Exacerbates the Progression of Cardiac Fibrosis in High-Fat-Fed Apolipoprotein E Knockout Mice via Endothelial-Mesenchymal Transition
  Tekijä: Ma, Kun Ling, et al.
  Julkaistu: (2013)
• Reduced VEGF production, angiogenesis, and vascular regrowth contribute to the antitumor properties of dual mTORC1/mTORC2 inhibitors
  Tekijä: Falcon, Beverly L., et al.
  Julkaistu: (2011)