Exogenous Administration of a Recombinant Variant of TWEAK Impairs Healing after Myocardial Infarction by Aggravation of Inflammation

BACKGROUND: Tumor necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor fibroblast growth factor-inducible 14 (Fn14) are upregulated after myocardial infarction (MI) in both humans and mice. They modulate inflammation and the extracellular matrix, and could therefore be important f...

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Autors principals: Pachel, Christina, Mathes, Denise, Bayer, Barbara, Dienesch, Charlotte, Wangorsch, Gaby, Heitzmann, Wolfram, Lang, Isabell, Ardehali, Hossein, Ertl, Georg, Dandekar, Thomas, Wajant, Harald, Frantz, Stefan
Format: Artigo
Idioma:Inglês
Publicat: Public Library of Science 2013
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Research Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3823964/
https://ncbi.nlm.nih.gov/pubmed/24244389
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0078938
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