Mesenchyme-specific Knockout of ESET Histone Methyltransferase Causes Ectopic Hypertrophy and Terminal Differentiation of Articular Chondrocytes

The exact molecular mechanisms governing articular chondrocytes remain unknown in skeletal biology. In this study, we have found that ESET (an ERG-associated protein with a SET domain, also called SETDB1) histone methyltransferase is expressed in articular cartilage. To test whether ESET regulates a...

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Autori principali: Lawson, Kevin A., Teteak, Colin J., Zou, Junhui, Hacquebord, Jacques, Ghatan, Andrew, Zielinska-Kwiatkowska, Anna, Fernandes, Russell J., Chansky, Howard A., Yang, Liu
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Biochemistry and Molecular Biology 2013
Soggetti:
Cell Biology
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3820852/
https://ncbi.nlm.nih.gov/pubmed/24056368
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M113.473827
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