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The Complement Anaphylatoxin Receptors Are Not Required for the Development of Experimental Autoimmune Uveitis
To determine if complement anaphylatoxin-mediated inflammation contributes to the development and progression of experimental autoimmune uveitis (EAU), we induced disease in wild type and complement anaphylatoxin receptor-deficient mice (C3a receptor(−/−), C5a receptor(−/−) and C3aR(−/−)/C5aR(−/−))...
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| Huvudupphovsmän: | , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
2013
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3818408/ https://ncbi.nlm.nih.gov/pubmed/24035596 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jneuroim.2013.08.010 |
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