Diversity and Similarity of Motor Function and Cross-Bridge Kinetics in Papillary Muscles of Transgenic Mice Carrying Myosin Regulatory Light Chain Mutations D166V and R58Q

Samankaltaisia teoksia

• Characterizations of myosin essential light chain’s N-terminal truncation mutant Δ43 in transgenic mouse papillary muscles by using tension transients in response to sinusoidal length alterations
  Tekijä: Wang, Li, et al.
  Julkaistu: (2013)
• Malignant familial hypertrophic cardiomyopathy D166V mutation in the ventricular myosin regulatory light chain causes profound effects in skinned and intact papillary muscle fibers from transgenic mice
  Tekijä: Kerrick, W. Glenn L., et al.
  Julkaistu: (2009)
• Impact of familial hypertrophic cardiomyopathy-linked mutations in the NH(2) terminus of the RLC on β-myosin cross-bridge mechanics
  Tekijä: Farman, Gerrie P., et al.
  Julkaistu: (2014)
• Myosin regulatory light chain mutation found in hypertrophic cardiomyopathy patients increases isometric force production in transgenic mice
  Tekijä: Kazmierczak, Katarzyna, et al.
  Julkaistu: (2012)
• The effect of myosin RLC phosphorylation in normal and cardiomyopathic mouse hearts
  Tekijä: Muthu, Priya, et al.
  Julkaistu: (2012)