The loss of sustained Ca(2+) signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

Lignende værker

• Changes in Ca(2+) Signaling and Nitric Oxide Output by Human Umbilical Vein Endothelium in Diabetic and Gestational Diabetic Pregnancies
  af: Anaya, Heather A., et al.
  Udgivet: (2015)
• Pregnancy Enhances Sustained Ca(2+) Bursts and Endothelial Nitric Oxide Synthase Activation in Ovine Uterine Artery Endothelial Cells Through Increased Connexin 43 Function
  af: Yi, Fu-Xian, et al.
  Udgivet: (2009)
• Positive versus negative effects of VEGF(165) on Ca(2+) signaling and NO production in human endothelial cells
  af: Boeldt, Derek S., et al.
  Udgivet: (2017)
• TNF-alpha Inhibits Pregnancy-Adapted Ca(2+) Signaling in Uterine Artery Endothelial Cells
  af: Ampey, Amanda C, et al.
  Udgivet: (2019)
• Phosphorylation of Ser-279/282 and Tyr-265 Positions on Cx43 as Possible Mediators of VEGF-165 Inhibition of Pregnancy-Adapted Ca2+ Burst Function in Ovine Uterine Artery Endothelial Cells
  af: Boeldt, Derek S, et al.
  Udgivet: (2015)