Endogenous Interleukin-1β in Neuropathic Rats Enhances Glutamate Release from the Primary Afferents in the Spinal Dorsal Horn through Coupling with Presynaptic N-Methyl-d-aspartic Acid Receptors

Míreanna Comhchosúla

• Molecular Mechanism of Neuropathic Pain♦: Endogenous Interleukin-1β in Neuropathic Rats Enhances Glutamate Release from the Primary Afferents in the Spinal Dorsal Horn through Coupling with Presynaptic N-Methyl-d-aspartic Acid Receptors
  Foilsithe: (2013)
• Endogenous activation of presynaptic NMDA receptors enhances glutamate release from the primary afferents in the spinal dorsal horn in a rat model of neuropathic pain
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  Foilsithe: (2013)
• Tachykinins and calcitonin gene-related peptide enhance release of endogenous glutamate and aspartate from the rat spinal dorsal horn slice
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  Foilsithe: (1990)
• Comparison of primary afferent and glutamate excitation of neurons in the mammalian spinal dorsal horn
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  Foilsithe: (1988)
• Evidence for presynaptic N-methyl-D-aspartate autoreceptors in the spinal cord dorsal horn.
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  Foilsithe: (1994)