Endogenous Interleukin-1β in Neuropathic Rats Enhances Glutamate Release from the Primary Afferents in the Spinal Dorsal Horn through Coupling with Presynaptic N-Methyl-d-aspartic Acid Receptors

Ítems similars

• Molecular Mechanism of Neuropathic Pain♦: Endogenous Interleukin-1β in Neuropathic Rats Enhances Glutamate Release from the Primary Afferents in the Spinal Dorsal Horn through Coupling with Presynaptic N-Methyl-d-aspartic Acid Receptors
  Publicat: (2013)
• Endogenous activation of presynaptic NMDA receptors enhances glutamate release from the primary afferents in the spinal dorsal horn in a rat model of neuropathic pain
  per: Yan, Xisheng, et al.
  Publicat: (2013)
• Tachykinins and calcitonin gene-related peptide enhance release of endogenous glutamate and aspartate from the rat spinal dorsal horn slice
  per: Kangrga, I, et al.
  Publicat: (1990)
• Comparison of primary afferent and glutamate excitation of neurons in the mammalian spinal dorsal horn
  per: Schneider, SP, et al.
  Publicat: (1988)
• Evidence for presynaptic N-methyl-D-aspartate autoreceptors in the spinal cord dorsal horn.
  per: Liu, H, et al.
  Publicat: (1994)