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The absence of FcγRIII results in increased pro-inflammatory response in FcγRIII-KO cardiac recipients
BACKGROUND: Alloantibody can contribute significantly to rejection of heart transplants by activation of complement and interactions with a variety of effector cells, including macrophages and monocytes through activating FcγRI, FcγRIII, FcγRIV, the inhibitory FcγRIIB and complement receptors. These...
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| Autors principals: | , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2013
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3795861/ https://ncbi.nlm.nih.gov/pubmed/23903009 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/TP.0b013e31829c2455 |
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