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Doubling down on PKC benefits allogeneic BMT
In this issue of Blood, using genetically deficient donor T cells or posttransplant pharmacologic inhibition, Haarberg et al have conclusively demonstrated that protein kinase C (PKC)-θ and PKC-α each contribute to alloreactivity after experimental allogeneic bone marrow transplantation (BMT).(1)
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| Hlavní autor: | |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society of Hematology
2013
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3790501/ https://ncbi.nlm.nih.gov/pubmed/24092926 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2013-08-519900 |
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