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Doubling down on PKC benefits allogeneic BMT

In this issue of Blood, using genetically deficient donor T cells or posttransplant pharmacologic inhibition, Haarberg et al have conclusively demonstrated that protein kinase C (PKC)-θ and PKC-α each contribute to alloreactivity after experimental allogeneic bone marrow transplantation (BMT).(1)

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Detalhes bibliográficos
Autor principal: Fowler, Daniel H.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society of Hematology 2013
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3790501/
https://ncbi.nlm.nih.gov/pubmed/24092926
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2013-08-519900
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