Eccentric contractions increase the phosphorylation of tuberous sclerosis complex-2 (TSC2) and alter the targeting of TSC2 and the mechanistic target of rapamycin to the lysosome

Eitemau Tebyg

• Identification of mechanically regulated phosphorylation sites on tuberin (TSC2) that control mechanistic target of rapamycin (mTOR) signaling
  gan: Jacobs, Brittany L., et al.
  Cyhoeddwyd: (2017)
• The role of phosphoinositide 3-kinase and phosphatidic acid in the regulation of mammalian target of rapamycin following eccentric contractions
  gan: O’Neil, T K, et al.
  Cyhoeddwyd: (2009)
• The Mechanical Activation of mTOR Signaling: An Emerging Role for Late Endosome/Lysosomal Targeting
  gan: Jacobs, Brittany L., et al.
  Cyhoeddwyd: (2013)
• Hepatitis C virus inhibits AKT-tuberous sclerosis complex (TSC), the mechanistic target of rapamycin (MTOR) pathway, through endoplasmic reticulum stress to induce autophagy
  gan: Huang, He, et al.
  Cyhoeddwyd: (2013)
• Insulinoma and Tuberous Sclerosis: A Possible Mechanistic Target of Rapamycin (mTOR) Pathway Abnormality?
  gan: Kang, Michael Yoon, et al.
  Cyhoeddwyd: (2017)