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Ethanol Metabolism Modifies Hepatic Protein Acylation in Mice

Mitochondrial protein acetylation increases in response to chronic ethanol ingestion in mice, and is thought to reduce mitochondrial function and contribute to the pathogenesis of alcoholic liver disease. The mitochondrial deacetylase SIRT3 regulates the acetylation status of several mitochondrial p...

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Bibliografiset tiedot
Päätekijät: Fritz, Kristofer S., Green, Michelle F., Petersen, Dennis R., Hirschey, Matthew D.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2013
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3779192/
https://ncbi.nlm.nih.gov/pubmed/24073283
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0075868
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