Human cytomegalovirus UL97 kinase prevents the deposition of mutant protein aggregates in cellular models of Huntington's disease and Ataxia

The presence of aggregates of abnormally expanded polyglutamine (polyQ)-containing proteins are a pathological hallmark of a number of neurodegenerative diseases including Huntington’s disease (HD) and spinocerebellar ataxia-3 (SCA3). Previous studies in cellular, Drosophila, and mouse models of HD...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Tower, Cristy, Fu, Lianwu, Gill, Rachel, Prichard, Mark, Lesort, Mathieu, Sztul, Elizabeth
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 2010
Gaiak:
Article
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC3771345/
https://ncbi.nlm.nih.gov/pubmed/20732421
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2010.08.013
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