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Yersinia pestis Subverts the Dermal Neutrophil Response in a Mouse Model of Bubonic Plague

The majority of human Yersinia pestis infections result from introduction of bacteria into the skin by the bite of an infected flea. Once in the dermis, Y. pestis can evade the host’s innate immune response and subsequently disseminate to the draining lymph node (dLN). There, the pathogen replicates...

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Detalhes bibliográficos
Main Authors: Shannon, Jeffrey G., Hasenkrug, Aaron M., Dorward, David W., Nair, Vinod, Carmody, Aaron B., Hinnebusch, B. Joseph
Formato: Artigo
Idioma:Inglês
Publicado em: American Society of Microbiology 2013
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3760243/
https://ncbi.nlm.nih.gov/pubmed/23982068
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/mBio.00170-13
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