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Amyloid-beta(1–42) reduces neuronal excitability in mouse dentate gyrus
Amyloid-β (Aβ) is causally implicated in Alzheimer’s disease and neuroplasticity failure has acquired validity as a possible mechanism of early AD pathogenesis. We have previously demonstrated that oligomeric Aβ (1–42) inhibits LTP in the dentate gyrus of rat hippocampal slices. We now show, using w...
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| 主要な著者: | , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2006
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3752836/ https://ncbi.nlm.nih.gov/pubmed/16765515 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neulet.2006.04.065 |
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