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Systemic Lack of Canonical Histamine Receptor Signaling Results in Increased Resistance to Autoimmune Encephalomyelitis
Histamine (HA) is a key regulator of experimental allergic encephalomyelitis (EAE), the autoimmune model of multiple sclerosis (MS). HA exerts its effects through four known G protein coupled receptors (GPCR): H(1), H(2), H(3), and H(4) (H(1-4)R). Using HR deficient mice, our lab has demonstrated th...
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| Autors principals: | , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2013
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3747232/ https://ncbi.nlm.nih.gov/pubmed/23772030 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1203137 |
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