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A mutant cholera toxin B subunit that binds GM1- ganglioside but lacks immunomodulatory or toxic activity
GM1-ganglioside receptor binding by the B subunit of cholera toxin (CtxB) is widely accepted to initiate toxin action by triggering uptake and delivery of the toxin A subunit into cells. More recently, GM1 binding by isolated CtxB, or the related B subunit of Escherichia coli heat-labile enterotoxin...
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| Main Authors: | , , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The National Academy of Sciences
2001
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC37471/ https://ncbi.nlm.nih.gov/pubmed/11447291 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.161273098 |
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