Quiescent complement in nonhuman primates during E coli Shiga toxin-induced hemolytic uremic syndrome and thrombotic microangiopathy

Enterohemorrhagic Escherichia coli (EHEC) produce ribosome-inactivating Shiga toxins (Stx1, Stx2) responsible for development of hemolytic uremic syndrome (HUS) and acute kidney injury (AKI). Some patients show complement activation during EHEC infection, raising the possibility of therapeutic targe...

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Bibliografski detalji
Glavni autori: Lee, Benjamin C., Mayer, Chad L., Leibowitz, Caitlin S., Stearns-Kurosawa, D. J., Kurosawa, Shinichiro
Format: Artigo
Jezik:Inglês
Izdano: American Society of Hematology 2013
Teme:
Platelets and Thrombopoiesis
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3731933/
https://ncbi.nlm.nih.gov/pubmed/23733336
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2013-03-490060
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