Quiescent complement in nonhuman primates during E coli Shiga toxin-induced hemolytic uremic syndrome and thrombotic microangiopathy

Enterohemorrhagic Escherichia coli (EHEC) produce ribosome-inactivating Shiga toxins (Stx1, Stx2) responsible for development of hemolytic uremic syndrome (HUS) and acute kidney injury (AKI). Some patients show complement activation during EHEC infection, raising the possibility of therapeutic targe...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Lee, Benjamin C., Mayer, Chad L., Leibowitz, Caitlin S., Stearns-Kurosawa, D. J., Kurosawa, Shinichiro
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society of Hematology 2013
Aiheet:
Platelets and Thrombopoiesis
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3731933/
https://ncbi.nlm.nih.gov/pubmed/23733336
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2013-03-490060
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