Mechanisms of HIV Entry into the CNS: Increased Sensitivity of HIV Infected CD14(+)CD16(+) Monocytes to CCL2 and Key Roles of CCR2, JAM-A, and ALCAM in Diapedesis

Ítems similars

• JAM-A and ALCAM are therapeutic targets to inhibit diapedesis across the BBB of CD14(+)CD16(+) monocytes in HIV-infected individuals
  per: Williams, Dionna W., et al.
  Publicat: (2015)
• Characterization and function of the human macrophage dopaminergic system: implications for CNS disease and drug abuse
  per: Gaskill, Peter J, et al.
  Publicat: (2012)
• Dopamine Increases CD14(+)CD16(+) Monocyte Transmigration Across the Blood Brain Barrier: Implications for Substance Abuse and HIV Neuropathogenesis
  per: Calderon, Tina M., et al.
  Publicat: (2017)
• CCR2 on CD14(+)CD16(+) monocytes is a biomarker of HIV-associated neurocognitive disorders
  per: Williams, Dionna W., et al.
  Publicat: (2014)
• CCL2/Monocyte Chemoattractant Protein-1 Mediates Enhanced Transmigration of Human Immunodeficiency Virus (HIV)-Infected Leukocytes across the Blood–Brain Barrier: A Potential Mechanism of HIV–CNS Invasion and NeuroAIDS
  per: Eugenin, Eliseo A., et al.
  Publicat: (2006)